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The treatment with the conditioned medium of endometriotic mesenchymal stem cells (Ecto-MSC) significantly increased the expression of genes involved in fibrogenesis in ECSCs via the Wnt/β-catenin pathway by paracrine production of TGF-β1 and Wnt1 [43], activating proliferation, invasion, and contraction of the collagen gel, thus supporting that Ecto-MSCs play a key role in the development of OMA (Fig