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Research shows that NNMT overexpression in adipose tissue creates a metabolic environment that favors lipid accumulation by depleting the very cofactors needed for efficient fatty acid oxidation

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Aging reduces NAD+ synthesis pathways while increasing consumption by enzymes like CD38 and PARPs, activated by inflammation and damage.[13][15] CD38, an immune-related enzyme, ramps up with age, breaking down NAD+ precursors